BGS 0013 desynapsis des4
Previous nomenclature and gene symbolization: None.
Inheritance: Monofactorial reces6ive (2,3). Located on chromosome 1 (1).
Description: The chromosomes are paired during pachytene and undergo desynapsis during diplotene. The degree of desynaps is is 3.3 + 2.2 ranging from 7 ring bivalents (d = O) to 3 rod bivalents plus 8 univalents (d = 11). Many univalents split longitudinally during anaphase I. Lagging chromosomes and micronuclei are frequently observed at telophase I. Microspore quartets contain an average of 1.0 micronuclei per quartet with a range of O to 10. Ovule fertility is about 18% (1).
Origin of mutant: Spontaneous occurrence in the cultivar Betzes C.I. 6398 (2,3).
Mutational events:
des4d spontaneous occurrence in the cultivar Betzes C.I.6398 (2,3).
des4h spontaneous occurrence in the cultivar Betzes C.I.6398 (2,3).
des4z, de64aa, des4ab, des4ac, des4ad, des4ae, des4af and des4ag spontaneous
occurrence in the cultivar Klages C.I. 15487 (4).
Mutant used for description and seed stock: des4d in Betzes C.I. 6398.
References:
1. Hernandez-Soriano, J. M. 1973. M.S. Thesis. University of Arizona,
Tucson.
2. Ramage, R. T. and J. M. Hernandez-Soriano. 1971. Barley Genetics
Newsletter 1: 38.
3. Ramage, R. T. and J. M. Hernandez-Soriano. 1972. Barley Genetics
Newsletter 2: 65-68.
4. Hernandez-Soriano, J. M. and R. T. Ramage. 1974. Barley Genetics
Newsletter 4: 123-125.
Prepared: J. M. Hernandez-Soriano, R. T. Ramage and R. F. Eslick. 1972.
Revised: R. T. Ramage and J. F. Scheuring, 1975.
BGS 0020 Resistant to Barley Yellow Mosaic Virus Rym2b
Previous nomenclature and symbolization: Resistance to BYMV, Ym2Ym2
Inheritance: Partially dominant, linked with naked kernel gene on chromosome l at a distance of 31.4 crossover units from naked kernel gene
Description: Resistance to barley yellow mosaic virus
Origin of mutant: Natural occurrence. in Mihori Hadaka 3.
Mutational events: Rym2b in Mihori Hadaka 3
Mutant used for description and seed stock: Rym2b in Mihori Hadaka 3
References:
l. Takahashi, Ryuhei, I. Hayashi, T. Inouye, I. Mortya and C. Hirao 1973. Berichte des Ohara Institute fur Landwirtschaftliche Biologie. Bd XVI, Heft. 1, S. 1-17
Prepared: R. G. Timian
BGS 0021 Resistant to Ustilago nuda (Jens.) Rostr. (Nuda Loose Smut) Run1a
Previous nomenclature and gene symbolization: Un (2)
Inheritance: Monfactorial dominance (1)
Located on Chromosome 1 (4)
Description: A major gene for resistance to "non-Trebi-attacking" U. nuda cultures (3,5) and to cultures capable of infecting the "Jet" type of resistance (6). Linked to a single dominant gene for resistance to stem rust. Puccinia graminis tritici Eriks. and Henn, in Valentine, CI 7242, with a recombination value of 2.04 + 0.45%, and with starch type with a recombination value of 12.74 + 1.71% (4).
Origin of mutant: Natural occurrence in Trebi, CI 936
Mutational events: Runla in Trebi
Mutant used for description and seed stock: Run1a in Trebi, CI 936
References:
1. Livingston, J. E. 1942. Phytopathology 32: 451-466.
2. Robertson, D. W. et al. 1947. J. Ame. Soc. Agron. 39: 464-473.
3. Schaller, C. 'S. 1949. Phytopathology 39: 959-979.
4. Shands, R. G. 1964. Phytopathology 54 #3: 308-316.
5. Skoropad, W. P. and L. P. V. Johnson. 1952. Can. J. Botany 30: 525-536.
6. Thomas, P. L. 1974. Can. J. Plant Sci. 54: 453-456.
Prepared: D. R. Metcalfe and P. L. Thomas. 1974.
BGS 0022 Resistant to Schizaphis graminum Rondani (Green bug) Rsg,,a
Previous nomenclature and symbolization: Grb (4)
Inheritance: Monofactorial dominance (1,2)
Chromosome 1 (3)
Description: Resistance in Omugi, CI 5144, Derhent, CI 5008, and Kearney, CI 7580, Conditioned by same gene or closely linked genes (2).
Origin of mutant: Natural occurrence in Omugi, Derfent and Kearney.
Mutational events: Rsg,,a in Omugi, Derbent and Kearney.
Mutant used for description and seed stock: Rsg,,a in Omugi, CI 5144
References:
1. Gardenhire, James H. 1965. Crop Science 5: 28-29.
2. Gardenhire, James H. and Harvey L. Chada. 1961. Crop Science 1:
349-352.
3. Gardenhire, J. H., N. A. Tuleen, and K. W. Stewart. 1973. Crop Science
13: 684-685.
4. Robertson, D. W., G. A. Wiebe and R. G. Shands. 1955. Agron. Jour.
47: 418-425.
Prepared: J. G. Moseman
BGS 0070 Resistant to Puccinia hordei Otth. (Barley Leaf Rust) Rph1a
Previous nomenclature and symbolization: Pa (2); Pa1 (4); C (1)
Inheritance: Monfactorial dominance under some circumstances may be partially dominant. Located on chromosome 2 (3)
Description: Seedling reaction type On - 1c with race 4 culture 57-19 (1); Heterozygotes range from type 1 to type 3, depending on parents. Responses will vary for homo- and heterozygote with different low virulence rust cultures.
Origin of mutant: Natural occurrence in Oderbrucker, CI 940; Speciale, CI 7536; and Sudan, CI 6489
Mutational events: Rph1a in Oderbrucker, Speciale, Sudan of the leaf rust differentials (1,2). There may be several alleles but genetic tests for allelic series are lacking.
Mutant used for description and seed stock: Rph1a in Oderbrucker, CI 940 (2,4)
References:
1. Roane, C. W. 1962. Phytopathology 52: 1288-1295.
2. Roane, C. W. and T. M. Starling. 1967. Phytopathology 57: 66-68.
3. Tuleen, N. A. and M. E. McDaniel. 1972. BNL 15: 106-107.
4. Watson, I. A. and F. C. Butler. 1948. Proc. Linnean Soc. N. S. Wales
72: 379-386.
Prepared: C. W. Roane
BGS 0121 Resistant to Puccinia hordei Otth. ( Barley Leaf Rust) Rph3c
Previous nomenclature and symbolization: Pa1 (1); Pa3 (2)
Inheritance: Monofactorial dominance (3,4)
May be linked with Rph5e (3,4) on Chromosome 3
Description: Seedling reaction type O - Oc with race
4, culture 57-19 (2)
Completely dominant.
Origin of mutant: Natural occurrence in Estate, CI 3410
Mutational events: Rph3c in Estate
Mutant used for description and seed stock: Rph3c in Estate, CI 3410 (1,2)
References:
1. Henderson, M. T. 1945. Ph. D. Dissertation. Univ. of Minn.
2. Roane, C. W. and T. M. Starling. 1967. Phytopathology 57: 66-68.
3. Roane, C. W. and T. M. Starling. 1970. Phytopathology 60: 788-790.
4. Tuleen, N. A. and M. E. McDaniel. 1972. BNL 15: 106-107.
Prepared: C. W. Roane
BGS 0122 Resistant to Puccinia hordei Otth. (Barley Leaf Rust) Rph5e
Previous nomenclature and symbolization: Pa5 (1,3,4) ; B (2)
Inheritance: Monfactorial dominance (2)
Located on chromosome 3 (5). Linked with Rph3c (4) and Rph7g (1)
Description: Seedling reaction with culture 57-19 of race 4, type On. Usually a necrotic lesion with brown pigment at the margin.
Origin of mutant: Natural occurrence in Quinn, CI 1024 (4), and Cebada Capa, CI 6193, (1,2,3,4)
Mutational events: Rph5e in Quinn in combination with Rph2b and in Cebada Capa (1,2,3,4)
Mutant used for description and seed stock: Rph5e Cebada Capa, CI 6193 (4)
References:
1. Frecha, J. H. 1970. Bol. Genet. Inst. Fitotec., Castelar 7: 1-8.
2. Roane, C. W. 1962. Phytopathology 52: 1288-1295.
3. Roane, C. W. and T. M. Starling. 1967. Phytopathology 57:
4. Roane, C. W. and T. M. Starling. 1970. Phytopathology 60: 788-790.
5. Tuleen, N. A. and M. E. McDaniel. 1972. BNL 15: 106-107.
Prepared: C. W. Roane
BGS 0123 Resistant to Barley Yellow Dwarf Virus (BYDV) Ryd2b
Previous nomenclature and symbolization: Resistant to BYDV Yd2 (1,2)
Inheritance: Monofactorial incomplete dominance (1,2)
Located on chromosome 3
Description: Intermediate resistance (tolerance to BYDV; limits expression of discoloration and dwarfing
Origin of mutant: Natural occurrence in four barleys from Ethiopia; CI 1227, CI 1237, CI 2376 and CI 14119
Mutational events: Ryd2b in CI 1227, CI 1237, CI 2376 and CI 14119
Mutant used for description and seed stock: Ryd2b in CI 2376
References:
1. Damsteegt, V. D., and G. W. Bruehl. 1964. Phytopath 54: 219-224.
2. Rasmusson, D. C. and C. W. Schaller. 1959. Agron. J. 51: 661-664.
3. Schaller, C. W., C. O. Qualset, and J. N. Rutger. 1964. Crop Sci.
4: 544-548.
Prepared: C. W. Schaller
BGS 0162 Resistant to Erysiphe graminis (DC) Merat hordei Em. Marchal (Powdery Mildew) Reg2ac
Previous nomenclature and symbolization: Mildew resistance G (1); Mlg (2); JMlg (3)
Inheritance: Monofactorial incomplete dominance (2,3,4)
Located on Chromosome 4. (3,4)
Description: Isogenic susceptible line derived by crossing Goldfoil, CI 928, with Manchuria, CI 2330, then backcrossing 3 times to Manchuria followed by selfing 14 generations. (4).
Origin of mutant: Natural occurrence in Goldfoil
Mutational events: Reg2ac in Goldfoil
Mutant used for description and seed stock: Reg2ac in Goldfoil/4*(F14) Man. CI 16139
References:
1. Briggs, F. N. and G. L. Barry. 1937. Zeits. Ziecht. A, 22: 75-80.
2. Briggs, F. N. and E. H. Stanford. 1938. Jour. of Genetics 37: 107-117.
3. Hiura, U. 1960. Ber. Ohara Inst. Landw. Biol. Okayama Univ. 11:
235-300.
4. Moseman, J. G. 1972. Crop Science 12: 681-682.
Prepared: U. Hiura, 1971, BGN 2:187
Revised: J.G. Moseman, 1976
BGS 0167 Resistant to Barley Yellow Mosaic Virus Rym1a
Previous nomenclature and symbolization: Resistant to BYMV, Ym Ym
Inheritance: Partially dominant, linked with hooded awn gene on chromosome 4 with 29.37% recombination (1)
Description: Resistant to barley yellow mosaic virus
Origin of mutant: Natural occurrence in Mokusekko 3
Mutational events: Rym1a in Mokusekko 3
Mutant used for description and seed stock: Rym1a in Mokusekko 3
References:
l. Takahashi, Ryuhei, I. Hayashi, T. Inouye, I. Moriya and C. Hirao.
1973. Berichte des Ohara Institute fur landwirtschaftliche Biologie. Bd.
XVI, Heft 1, S. 1-17.
Prepared: R. G. Timian
BGS 0209 Resistant to Erysiphe graminis (DC) Merat hordei Em. Marchal (Powdery Mildew) Reg4ae
Previous nomenclature and symbolization: Resistant to E. graminia hordei Ml-k, (1) JMlk, (2). (BGN 2:193)
Inheritance: Monofactorial dominance. (1,2,7)
Located on Chromosome 5. (2,7,9)
Description: Homoyzgous resistant 1-2, 2 type infection difficult to distinguish from heterozygous plants. Approximately 8 and 22 percentage recombinations from genes Reg1a and Rph4d respectively (4,5,8,9) inherited independently of gene Reg5af and Ml-at (2,3,4,9) and at or near same locus as gene JMl-nz (2,3). Isogenic resistant lines derived by crossing Kwan, CI 1016, with Manchuria, CI 2330, then backcrossing 3 times to Manchuria followed by selfing 12 generations. (7).
Origin of mutant: Natural occurrence in variety Kwan, CI 1016
Mutational events: Reg4ae in Kwan.
Mutant used for description and seed stock: Reg4ae in Kwan/4*(F12) Man CI 16143.
1. Briggs, F. N. and E. H. Stanford. 1938. Jour.of Genetics 37: 107-117.
2. Hiura, U. 1960. Ber. Ohara Inst. Landwirtsch. Biol. Okayama Univ.
11: 235-300.
3. Jensen, J. and J. H. Jørgensen. 1975a. Hereditas (In press).
4. Jensen, J. and J. H. Jørgensen. 1975b. Hereditas (In press).
5. McDaniel, M. E. and B. R. Hothcock. 1969. Crop Science 9:822.
6. Moseman, J. G. 1966. Ann. Rev. Phytopathology 4: 269-290.
7. Moseman, J. G, 1972. Crop Science 12: 681-682.
8. Moseman, J. G. and D. A. Reid. 1961. Crop Science 1: 425-427.
9. Schaller, C. W. and F. N. Briggs. 1955. Genetics 40: 421-428.
Prepared: U. Hiura 1971, BGN 2:193
Revised: J.G. Moseman, 1975
BGS 0216 Resistant to Erysiphe graminis (DC) Merat hordei Em. Marchal (Powdery Mildew) Reg1al Reg1aa
Previous nomenclature and symbolization: Resistant to E. graminis hordei Ml-a, (1,2) or Jmlasn, (3)
Inheritance: One dominance gene at or near Reg1 locus on Chromosome 5. (1,2,3,6,8). Second dominance gene at Reg1 locus (10). Reg1 locus approximately 8,16 and 40 percentage recombinations from genes Reg4ae, Rph4d and Reg5af respectively, and inherited independently of genes B, at, and trd (4,5,9)
Description: Homozygous resistant O type infection conditioned by Reg1al can be distinguished from heterozygous plants. Homozygous resistant 1-2 type infection conditioned by gene Reg1aa cannot be distinguished from heterozygous plants. Isogenic resistant line derived by crossing Algerian, CI 1179, with Manchuria, CI 2330, then backcrossing 3 times to Manchuria followed by selfing 14 generations. (7).
Origin of mutant: Natural occurrence in variety Algerian, CI 1179
Mutational events: Reg1al; Reg1aa in Algerian
Mutant used for description and seed stock: Reg1al; Reg1aa in Algerian/4*(F14) Man CI 16137
References:
1. Briggs, F. N. and E. H. Stanford. 1938. Jour. of Genetics 37: 107-117.
2. Favret, E. A. 1960. Hereditas 46: 20-28.
3. Hiura, U. 1960. Ber. Ohara Inst. Landwirtsch. Biol. Okayama Univ.
11:235-300
4. Jensen, J. and J. H. Jørgensen. 1975a. Hereditas (In press).
5. Jensen, J. and J. H. Jørgensen. 1975b. Hereditas (In press).
6. Moseman, J. G. 1966. Ann. Rev. Phytopathology 4: 269-290.
7. Moseman, J. G. 1972. Crop Science 12: 681-682.
8. Schaller, C. W. and F. N. Briggs. 1955. Genetics 40:421-428.
9. Scholz, F. and I. Nover. 1967. Kulturplanze 15: 243-254.
10. Moseman, J. G. 2nd Gene at Regl Locus Effective Against Culture
63-5. Unpublished data.
Prepared: J. G. Moseman. 1975
BGS 0217 Resistant to Erysiphe graminis (DC) Merat hordei Em. Marchal (Powdery Mildew) Reg5af
Previous nomenclature and symbolization: Resistant to E. graminis hordei Ml-p, (8,3) JMlp, (8,3) JMlp, (2)
Inheritance: Monofactorial dominance. (9,4)
Located on Chromosome 5. (1,3,5,6,7,8)
Description: Homozygous resistant 1-2 type infection cannot be distinguished from heterozygous plants. Gene effective against pathogenic strains of pathogen present in many countries, but not against those in Israel. Isogenic resistant line derived by crossing Psaknon, CI 6305 with Manchuria, CI 2330 then backerossing 3 times to Manchuria followed by selfing 14 generations. (4).
Origin of mutant: Natural occurrence in variety Psaknon, CI 6305.
Mutational events: Reg5af in Psaknon
Mutant used for description and seed stock: Reg5af in Psaknon/4* (F14) Man CI 16145
References:
Briggs, F. N. 1945. Genetics 30: 115-118.
Hiura, U. 1960. Ber. Ohara Inst. Landwirtsch. Biol. Okayama Univ. 11:235-300
Moseman, J. G. 1966. Ann. Rev. Phytopath. 4: 269-290.
Moseman, J. G. 1972. Crop Science 12: 681-682.
Moseman, J. G. and C. W. Schaller. 1960. Phytopathology 50: 736-741.
Moseman, J. G. and C. W. Schaller. 1962. Phytopathology 52: 529-533.
Schaller, C. W. and F. N. Briggs. 1955. Genetics 40: 421-428.
Scholz, F. and I. Nover. 1967. Kulturpflanze 15: 243-254.
Stanford, E. H. and F. N. Briggs. L940. G. Agr. Res. 61: 231-236.
Prepared: J. G. Moseman. 1975
BGS 0218 Resistant to Puccinia hordei Otth. (Barley Leaf Rust) Rph4d
Previous nomenclature and symbolization: Pa4 (3,4); D (3)
Inheritance: Monofactorial dominance (3)
Located on chromosome 5, 16% recombinations from gene Reg1al. (1,2)
Description: Seedling reaction with culture 57-19 of race 4, type On - 1c; heterozygotes may be type 2 or 3 under some circumstances but usually are 1c _ ln.
Origin of mutant: Natural occurrence in Gold, CI 1145; and Lechtaler, CI 6488
Mutational events: Rph4d in Gold and Lechtaler (3)
Mutant used for description and seed stock: Rph4d in Gold, CI 1145 (1,2)
References:
1. McDaniel, M. E, and B. R. Hathcock. 1969. Crop Sci. 9: 822.
2. Moseman, J. G. and D. A. Reid. 1961 Crop Sci. 1: 425-427.
3. Roane, C. W. 1962. Phytopathology 52: 1288-1295.
4. Roane, C. W. and T. M. Starling. 1967. Phytopathology 57: 66-68.
Prepared: C. W. Roane
BGS 0219 Resistant to Puccinia striiformis West (Stripe Rust) Rps4d
Previous nomenclature and symbolization: Resistant to Puccinia striiformis, Yr. 4, (2,3)
Inheritance: Monofactorial dominance (2,3)
Located on chromosome 5 (1)
Description: Causes '00' or 'O' reaction to Puccinia strtiformis race 23 (4), (2). Note - Does not give resistance to race 24 (2).
Origin of mutant: Natural occurrence in varieties Europa, Cambrinus, CI 13533 and Deba Abed (1,2,3)
Mutational events: Rps4d in Europa, Cambrinus, and Deba Abed, (1,2)
Mutant used for description and seed stock: Rps4d in Cambrinus, CI 13533 (1,2)
References:
1. Finch, R. A. et al (Unpublished).
2. Johnson, R. 1968. First International Congress of Plant Pathology
London 1968, p. 99
3. Moseman, J. G. 1971. Second International Barley Genetics Symposium,
Pullman, Washington, U.S.A. 1969. 535-543.
4. Straib, W. 1944. Zuchter 16, 64-68.
Prepared: R. Johnson and R. A. Finch
BGS 0257 smooth awns r,,e
Previous nomenclature and gene symbol: Rough vs. smooth awns R2 r2 (1) and (R5) (r5) (2)
Inheritance: Monofactorial recessive (1)
Linked to orange lemma (o) on chromosome 6 (1, 2)
Description: As compared with the strains with r for smooth awn, the new smooth awned mutant was characterized by somewhat fewer stigma hairs and reduced seed fertility, whereas this developed slightly more barbs in the central portion of its awns (2)
Origin of mutant: Induced mutant from a naked (n) "uzu" cultivar Akashinriki by EMS treatment
Mutational Events: r,,e in Akashinriki
Mutant used for description and seed stock: r,,e in O U M 195 Genotype n v uz k b O S R
References:
(1) Konishi, T. 1971. BGN 1: 25-26.
(2) Konishi, T. 1975. Nogaku Kenkyu 55: 53-66.
Prepared by: T. Konishi 1976
BGS 0258 dense spike l9
Previous nomenclature and gene symbol: la (1)
Inheritance: Monofactorial recessive
Located on chromosome 6 (1)
Description: Extremely dense spikes, easily distinguishable from the normal types grown in open field or greenhouse, even at dense planting.
Origin of mutant: EMS - induced dwarf mutants with a dense spike from the cultivar "Akashinriki".
Mutational Events: 5 independent mutational events.
Mutant used for description and seed stock: l9 in O U M 113 Genotype: n v uz k b O S R
References:
(1) Konishi, T. 1973. BGN 3: 31
Prepared by T. Konishi 1976
BGS 0259 alpha amylase Amy 1
Previous nomenclature and gene symbol:
Amy 1 Bi = Amy (2) = alpha- amyC (1)
Amy 1 Ke = amy (2) = alpha - amyC1(l)
Inheritance: Monofactorial codominant alleles (3)
Located on chromosome 6 distal to xn in the long arm (3)
Description: Electrophoretically defined alpha-amylase patterns. See description: Frydenberg et. al. (1969).
Origin of mutant: Natural, i.e. variants exist in cultivars.
Mutational Events: Spontaneous occurrence
Mutant used for description and seed stock: Amy 1 Ke: cultivar Kenia KVL 301 (1) (for seed stock)
Amy 1 Bi: cultivar Bido A. Hor. 3783/64 (1)
Stocks BGS-0251, o252, 0253, 0254, 0255 and 0354 contain the Amy 1
Bi allele.
References:
(1) Frydenberg, O., G. Nielsen and J. Sandfaer. 1969. A. Pflanzenzucht.
61: 201-215.
(2) Nielsen, G. and O Frydenberg. 1971. BGN 1: 34
(3) Nielsen, G. and O.Frydenberg. 1974. BGN 4: 53-54.
Prepared by: G. Nielsen and K.J. Kasha
BGS 0311 Shrunken endosperm, xenia sex 2b
Previous nomenclature and symbolization: None.
Inheritance: Monofactorial recessive expressing xenia (1).
Located on chromosome 7 (1).
Deseription: The phenotype is "thin" seed. Homozygous recessive seed grow normally for about 15 days after fertilization. Then, growth stops and the seed begin to shrivel. At maturity, shrunken endosperm are so reduced in size that they are barely visible within the lemma and palea. Shrunken endosperm seed are inviable. The mutant expresses xenia. Seed produced on heterozygous plants are found in a ratio of sbout 3 normal viable seed: 1 shrunken inviable seed (1).
Origin of mutant: Spontaneous occurrence in the balanced tertiary trisomic line 63-j-18-17. (1).
Mutational events: sex 2b spontaneous occurrence in the BTT line 63-J-18-17 (1).
Mutant used for description and seed stock: sex 2b in the BTT line 63-J-18-17. Seed stock is a tertiary trisomic for chromosome T27d balanced for both sex 2b and the male sterile allele msg2.
References:
Scheuring, J. F. and R. T. Ramage. A tertiary trisomic balanced for
both msg2 and sex 2. Barley Genetics Newsletter 6: 60-62.
Prepared: J. F. Scheuring and R. T. Ramage. 1975.
BGS 377 shrunken endosperm seg1
Previous nomenclature and symbolization: se 1 se 1 (prior 1976). (BGN 1:190)
Inheritance: Monofactorial recessive (1).
Located on chromosome 1 (1).
Description: The phenotype is "thin" seeds. The 100 seed weight of the mutant is about 33% of normal. Pollen mother cell meiosis, endosperm mitosis and fertility are normal. The mutant does not express xenia. Good stands of this mutant can be grown under field conditions if optimum environmental conditions prevail during germination and emergence (1). Associated with this gene is an increase in percent lysine in the protein.
Origin of mutant: Spontaneous occurrence in the cultivar Betzes C.I. 6398 (1).
Mutational events: seg1a in Betzes C.I. 6398, spontaneous (1).
Mutant used for description and seed stock: seg1a in Betzes C.I. 6398. Genotype: V b k N R S.
References:
(1) Jarvi, A. J. 1970. Ph.D. Thesis. Montana State University, Bozeman,
Montana.
(2) Jarvi, A. J. and R. F. Eslick. 1975. Crop Science 15:363-366.
Prepared: A. J. Jarvi and R. F. Eslick. 1970.
Revised: R.F. Eslick. 1976.
BGS 378 shrunken endosperm seg2
Previous nomenclature and symbolization: se 2 se 2 (prior 1976). (BGN 1:190)
Inheritance: Monofactorial recessive (1).
Located on chromosome 1 (1).
Description: The phenotype is "thin" seeds. The 100 seed weight of the mutant is about 15% of normal. Pollen mother cell meiosis, endosperm mitosis and fertility are normal. The mutant does not express xenia. There is little or no starch deposited in the seed that develops on a homozygous mutant plant. Seed from the homozygous mutant never produce plants under field conditions. Plants can be obtained from the shrunken endosperm seeds by germinating the seed on blotters with a 1O% sucrose solution. About 25% of the seeds germinated produce plants after transplanting the seedlings to soil when the coleoptile is about 3 cm in length (1).
Origin of mutant: Spontaneous occurrence in the cultivar Betzes C.I. 6398 (1).
Mutational events: seg2b in Betzes C.I. 6398, spontaneous (1).
Mutant used for description and seed stock: seg2b in Betzes C.I. 6398.
Genotype: V b k N R S.
The most generally acceptible method of reproducing this stock is with
the T16c seg2 seg2 balanced tertiary trisomic stock (BGN 1), available
from R. T. Ramage.
References:
(1) Jarvi, A. J. 1970. Ph.D. Thesis. Montana State University, Bozeman,
Montana.
(2) Jarvi, A. J. and R. F. Eslick, 1975. Crop Science 15:363-366.
(3) R. T. Ramage, BGN 1:74-80.
Prepared: A. J. Jarvi and R. F. Eslick. 1970.
Revised: R.F. Eslick. 1976.
BGS 379 shrunken endosperm seg3
Previous nomenclature and symbolization: se 3 se 3 (prior 1976). (BGN 1:191)
Inheritance: Monofactorial recessive (1).
Located on chromosome 3 (1).
Description: The phenotype is "thin" seed. The 100 seed weight of the mutant is about 33% of normal. Pollen mother cell meiosis, endosperm mitosis and feritlity are normal. The mutant does not express xenia. Generally good stands of this mutant can be obtained under field conditions (1).
Origin of mutant: Spontaneous occurrence in the cultivar Compana C.I. 5438 (1).
Mutational events: seg3c in Compana C.I. 5438, spontaneous (1).
Mutant used for description and seed stock: seg3c in Compana C.I. 5438. Genotype: V b k N R S.
References:
(1) Jarvi, A. J. 1970. Ph.D. Thesis. Montana State University,
Bozeman, Montana.
(2) Jarvi, A. J. and R. F. Eslick. 1975. Crop Science 15:363-366.
Prepared: A. J. Jarvi and R. F. Eslick. 1970.
Revised: R.F. Eslick. 1976.
BGS 380 shrunken endosperm seg4
Previous nomenclature and symbolization: se 4 se 4 (prior 1976). (BGN 1:192)
Inheritance: Monofactorial recessive (1).
Located on chromosome 1 (1).
Description: The phenotype is "thin" seeds accompanied by about 50% female sterility. The sterile florets appear to start development but abort before they reach half the length of the lemma. The 100 seed weight of the mutant is about 38% of normal. Pollen mother cell meiosis and endosperm mitosis are normal. The mutant does not express xenia. The mutant can be grown under field conditions but poor stands are obtained when less than optimum conditions prevail during germination and emergence (1) .
Origin of mutant: Spontaneous occurrence in the cultivar Compana, C.I. 5438 (1).
Mutational events: seg4d in Compana C.I. 5438, spontaneous (1).
Mutant used for description and seed stock: seg4d in Compana C.I. 5438. Genotype: V b k N R S.
References
(1) Jarvi, A. J. 1970. Ph.D. Thesis. Montana State University, Bozeman,
Montana.
(2) Jarvi, A. J. and R. F. Eslick. 1975. Crop Science 15:363-366.
Prepared: A. J. Jarvi and R. F. Eslick. 1970.
Revised: R.F. Eslick. 1976.
BGS 381 shrunken endosperm seg5
Previous nomenclature and symbolization: se 5 se 5 (prior 1976). (BGN 1:192)
Inheritance: Monofactorial recessive (l).
Located on chromosome 1 (1).
Description: The phenotype is "thin" seeds accompanied by about 80% female sterility. The female sterility appears to be due to aborted seed. The caryopsis of the mutant generally extends beyond the lemma and palea. The 100 seed weight of the mutant is quite variable, averaging about 50% of normal. Pollen mother cell meiosis and endosperm mitosis are normal. The mutant does not express xenia (1).
Origin of mutant: Spontaneous occurrence in a Sermo x Glacier line (1).
Mutational events: seg5 in a Sermo 17* Glacier line, spontaneous (1).
Mutant used for description and seed stock: seg5 in Sermo 17* Glacier line. Genotype: v b k N r s.
References:
(1) Jarvi, A. J. 1970. Ph.D. Thesis. Montana State University. Bozeman,
Montana.
(2) Jarvi, A. J. and R. F. Eslick. 1975. Crop Science 15:363-366.
Prepared: A. J. Jarvi and R. F. Eslick. 1970.
Revised: R.F. Eslick. 1975.
BGS 382 shrunken endosperm sex1
Previous nomenclature and symbolization: se 6 se 6 (prior 1976). (BGN 1:193)
Inheritance: Monofactorial recessive expressing xenia (1).
Located on chromosome 6 (1).
Description: After the hard dough stage, seeds of the mutant develop a depression in the center of the lemma which becomes progressively more distinct with maturity. The 100 seed weight of the mutant is about 75% of normal. Pollen mother cell meiosis, endosperm mitosis and fertility are normal. The mature endosperm of the mutant appears to be much harder than normal. The mutant expressed xenia permitting 1:2:1 classification in F2. Field establishment and growth are normal (1).
Origin of mutant: Spontaneous occurrence in the cultivar Compana C.I. 5438 (1).
Mutational events: sex6f in Compana C.I. 5438, spontaneous (l).
Mutant used for description and seed stock: sex6f in Compana C.I. 5438. Genotype: V b k N R S.
References:
(1) Jarvi, A. J. 1970. Ph.D. Thesis. Montana State University, Bozeman,
Montana.
(2) Jarvi, A. J. and R. F. Eslick. Crop Science 15:363-366.
Prepared: A. J. Jarvi and R. F. Eslick. 1970.
Revised: R.F. Eslick and E.A. Hockett. 1976.
BGS 0396 Shrunken endosperm seg6
Previous nomenclature and symbolization: se6 (BGN 5:114)
Inheritance: Monofactorial recessive. (1)
Description: The phenotype is "thin" seed. The expression of the character is affected by environment. Seed weights of 25, 50 and 75 per cent of normal were obtained from plants grown in the field in Arizona, in the field in Montana and in the greenhouse in Arizona. Pollen mother cell meiosis and fertility are normal. No difficulties have been encountered in establishing stands of this mutant under field conditions. (1)
Origin of mutant: Spontaneous occurrence in the cultivar Ingrid C.I.10083.
Mutational events: seg6f spontaneous occurrence in material of hybrid
origin
seg6g spontaneous occurrence in the cultivar Ingrid C.I.10083.
Mutant used for description and seed stock: seg6g in Ingrid C.I.10083.
Reference
(1) Ramage, R. T. and J. F. Scheuring. Shrunken endosperm mutants
seg6 and seg7. Barley Genetics Newsletter 6:
Prepared: R. T. Ramage and R. F. Eslick. 1974.
Revised: R. T. Ramage and J. F. Scheuring. 1975.
BGS 0397 Shrunken endosperm seg7
Previous nomenclature and symbolization: se7 (BGN 5:115)
Inheritance: Monofactorial recessive. (1)
Description: The phenotype is "thin" seed. The expression of the character is affected by environment. Seed weights of 40, 75 and 90 per cent of normal were obtained from plants grown in the field in Arizona, in the field in Montana and in the greenhouse in Arizona. Pollen mother cell meiosis and fertility are normal. No difficulties have been encountered in establishing stands of this mutant under field conditions. (1)
Origin of mutant: Spontaneous occurrence in the cultivar Ingrid C.I.10083.
Mutational events: seg7h spontaneous occurrence in the cultivar Ingrid C.I.10083.
Mutant used for description and seed stock: seg7h in Ingrid C.I.10083.
References
(1) Ramage, R. T. and J. F. Scheuring. Shrunken endosperm mutants seg6
and seg7. Barley Genetics Newsletter 6:
Prepared: R. T. Ramage and R. F. Eslick. 1974.
Revised: R. T. Ramage and J. F. Scheuring. 1975.
BGS 0452 Resistant to Erysiphe graminis (DC) Merat hordei Em. Marchal (Powdery Mildew) Reg3ad
Previous nomenclature and symbolization: Resistant to E. Graminis hordei Ml-h, (1) JMlh, (4)
Inheritance: Monofactorial dominance. (1,4)
Independent of linkage groups on Chromosomes 4 and 5. (2,3,4,6)
Description: Homozygous resistant 2 type infections cannot be distinguished from heterozygous plants. Isogenic lines derived by crossing Hanna, CI 906 with Manchuria, CI 2330, then backerossing 3 times to Manchuria followed by selfing 12 generations. (7).
Origin of mutant: Natural occurrence in Hanna, CI 906
Mutational events: Reg3ad in Hanna
Mutant used for description and seed stock: Reg3ad Hanna/4*(F12) Man CI 16141
References:
1. Briggs, P. N. 1935. Jour. Agrc. Res. 51: 245-250.
2. Briggs, F. N. 1945. Genetics 30: 115-118.
3. Briggs, F. N. 1937. Pflanzenzucht 22: 72-80.
4. Hiura, U. 1960. Ber. Ohara Inst. Landwirtsch. Biol. Okayama Univ.
11: 235-300.
5. Luig, N. H. , K. S. McWhirter and E. P. Baker. 1958. Proc. Linnean
Soc. N. S. Wales 83: 340-362.
6. Moseman, J. G. 1966. Ann. Rev. Phytopathology 4: 269-290.
7. Moseman, J. G. 1972. Crop Science 12: 681-682.
Prepared: J. G. Moseman. 1975